Sign inGet started
Paper · arXiv

Reward Valuation in Large Language Models: Causal Induction of Anhedonia

Recent frontier models mimic complex aspects of human cognition. Here we ask whether this alignment extends into reward valuation, which we assess in a mechanistic framework. Specifically, we use clinical tests that were developed to evaluate anhedonia in human subjects with major depressive disorders. Mechanistically, anhedonia is frequently associated with dysregulation in the Nucleus Accumbens (NAc) and the broader dopaminergic reward system. While neuroimaging has localized these deficits, establishing a causal link between NAc activity and specific behavioral symptoms remains a challenge. We use these ideas from neuroscience to functionally identify reward-anticipatory units in state...

AuthorsMelika Honarmand, Samin Mahdipour Aghabagher, Martin Schrimpf
Primary categorycs.LG
Published7 July 2026
Last updated at source30 September 2026
cs.LGq-bio.NC
Embed this entry
<iframe src="https://silytics.si/embed/item/79a7524e-8dad-4985-9ccc-74104f92b205" width="420" height="190" style="border:0" loading="lazy" title="Silytics entry"></iframe>

Free, no tracking. More about feeds and embeds.

Silytics shows public metadata from arXiv and links to the original. Information may be incomplete or out of date. Last refreshed 17 October 2026. Licenses and terms of the original apply to the work itself. Not financial, legal or professional advice.